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Researchers at the University of California, Riverside have proposed a new explanation for how Alzheimer's disease may begin. Instead of being driven primarily by plaque buildup in the brain, the disease could start when one protein interferes with the normal function of another inside nerve cells.
For years, Alzheimer's research has largely centered on amyloid beta (a-beta), a protein that forms clumps in the brains of people with the disease. The idea gained...
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